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Cite this DOI

10.46243/jst.2025.v10.i04.pp62-72 · Integrative Analysis of Oxidative Stress Biomarkers And Their ” Clinical correlates in Pediatric Type 1 Diabetes A MultiParametric Approach

APA (7th edition)

M Kishore Babu, T Pavani, D Divyasri, & P Krupa Rani (2025). Integrative Analysis of Oxidative Stress Biomarkers And Their ” Clinical correlates in Pediatric Type 1 Diabetes A MultiParametric Approach. *Journal of Science & Technology*, *10*(04), 62–72. https://doi.org/10.46243/jst.2025.v10.i04.pp62-72

⬇ text Italics are shown as *asterisks* in plain text — the journal or book title and the volume.

BibTeX

@article{mkishorebabu2025integrative,
  author    = {M Kishore Babu and T Pavani and D Divyasri and P Krupa Rani},
  title     = {{Integrative Analysis of Oxidative Stress Biomarkers And Their ” Clinical correlates in Pediatric Type 1 Diabetes A MultiParametric Approach}},
  journal   = {Journal of Science \& Technology},
  year      = {2025},
  month     = {apr},
  volume    = {10},
  number    = {04},
  pages     = {62--72},
  publisher = {Longman Publishers},
  issn      = {2456-5660},
  doi       = {10.46243/jst.2025.v10.i04.pp62-72},
  url       = {https://doi.org/10.46243/jst.2025.v10.i04.pp62-72},
  language  = {en},
  abstract  = {n this study, we used the neutrophil granulocytes functional activity model to examine oxidative stress indicators, antioxidant protection, lipid peroxidation, and T1D in children. According to research, children with type 1 diabetes show heightened lipoperoxidation and antioxidant defense system activation in response to reactive oxygen species (ROS) damage, which includes bidirectional alterations in non-enzymatic pathways. The development of tolerance to oxidative stress in children with compensated type 1 diabetes is indicated by the coordinated synthesis of reactive oxygen species and oxygen-dependent blood cell metabolism. In children with decompensated type 1 diabetes, it is indicated that the third stage of oxidative stress, which involves the depletion of insulin-producing β-cells, begins with a decrease in reactive oxygen species production, a decrease in the activation of the phagocyte oxygen-dependent metabolism, and incomplete phagocytosis mechanisms. The severity of metabolic problems in children with type 1 diabetes is influenced by the level of the antioxidant defense system's "respiratory explosion" of neutrophilic granulocytes.}
}

⬇ .bib

RIS (EndNote, Zotero, Mendeley)

TY  - JOUR
TI  - Integrative Analysis of Oxidative Stress Biomarkers And Their ” Clinical correlates in Pediatric Type 1 Diabetes A MultiParametric Approach
AU  - M Kishore Babu
AU  - T Pavani
AU  - D Divyasri
AU  - P Krupa Rani
JO  - Journal of Science & Technology
PY  - 2025
DA  - 2025/04/05/
VL  - 10
IS  - 04
SP  - 62
EP  - 72
PB  - Longman Publishers
SN  - 2456-5660
LA  - en
AB  - n this study, we used the neutrophil granulocytes functional activity model to examine oxidative stress indicators, antioxidant protection, lipid peroxidation, and T1D in children. According to research, children with type 1 diabetes show heightened lipoperoxidation and antioxidant defense system activation in response to reactive oxygen species (ROS) damage, which includes bidirectional alterations in non-enzymatic pathways. The development of tolerance to oxidative stress in children with compensated type 1 diabetes is indicated by the coordinated synthesis of reactive oxygen species and oxygen-dependent blood cell metabolism. In children with decompensated type 1 diabetes, it is indicated that the third stage of oxidative stress, which involves the depletion of insulin-producing β-cells, begins with a decrease in reactive oxygen species production, a decrease in the activation of the phagocyte oxygen-dependent metabolism, and incomplete phagocytosis mechanisms. The severity of metabolic problems in children with type 1 diabetes is influenced by the level of the antioxidant defense system's "respiratory explosion" of neutrophilic granulocytes.
DO  - 10.46243/jst.2025.v10.i04.pp62-72
UR  - https://doi.org/10.46243/jst.2025.v10.i04.pp62-72
ER  -

⬇ .ris

CSL-JSON

{
    "type": "article-journal",
    "id": "10.46243/jst.2025.v10.i04.pp62-72",
    "DOI": "10.46243/jst.2025.v10.i04.pp62-72",
    "URL": "https://doi.org/10.46243/jst.2025.v10.i04.pp62-72",
    "title": "Integrative Analysis of Oxidative Stress Biomarkers And Their ” Clinical correlates in Pediatric Type 1 Diabetes A MultiParametric Approach",
    "source": "Smart Scholars DOI Registry",
    "container-title": "Journal of Science & Technology",
    "author": [
        {
            "family": "M Kishore Babu"
        },
        {
            "family": "T Pavani"
        },
        {
            "family": "D Divyasri"
        },
        {
            "family": "P Krupa Rani"
        }
    ],
    "issued": {
        "date-parts": [
            [
                2025,
                4,
                5
            ]
        ]
    },
    "volume": "10",
    "issue": "04",
    "page": "62-72",
    "publisher": "Longman Publishers",
    "language": "en",
    "abstract": "n this study, we used the neutrophil granulocytes functional activity model to examine oxidative stress indicators, antioxidant protection, lipid peroxidation, and T1D in children. According to research, children with type 1 diabetes show heightened lipoperoxidation and antioxidant defense system activation in response to reactive oxygen species (ROS) damage, which includes bidirectional alterations in non-enzymatic pathways. The development of tolerance to oxidative stress in children with compensated type 1 diabetes is indicated by the coordinated synthesis of reactive oxygen species and oxygen-dependent blood cell metabolism. In children with decompensated type 1 diabetes, it is indicated that the third stage of oxidative stress, which involves the depletion of insulin-producing β-cells, begins with a decrease in reactive oxygen species production, a decrease in the activation of the phagocyte oxygen-dependent metabolism, and incomplete phagocytosis mechanisms. The severity of metabolic problems in children with type 1 diabetes is influenced by the level of the antioxidant defense system's \"respiratory explosion\" of neutrophilic granulocytes.",
    "ISSN": "2456-5660"
}

⬇ .json What citeproc and reference managers read; the DOI system hands it out for Accept: application/vnd.citationstyles.csl+json, and so does this registry's resolver.

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